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Showing 1 to 12 of 6108 entries
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GPER Signaling in Spermatogenesis and Testicular Tumors.

Frontiers in endocrinology

Chimento A, Sirianni R, Casaburi I, Pezzi V.
PMID: 24639669
Front Endocrinol (Lausanne). 2014 Mar 06;5:30. doi: 10.3389/fendo.2014.00030. eCollection 2014.

Estrogens play important roles in the regulation of testis development and spermatogenesis. Moreover, several evidences suggest that estrogen signaling can be involved in testicular tumorigenesis. The physiological effects of estrogen are mediated by the classical nuclear estrogen receptors ESR1...

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Chimento A, Sirianni R, Casaburi I, et al. GPER Signaling in Spermatogenesis and Testicular Tumors. Front Endocrinol (Lausanne). 2014;5:30doi: 10.3389/fendo.2014.00030.
Chimento, A., Sirianni, R., Casaburi, I., & Pezzi, V. (2014). GPER Signaling in Spermatogenesis and Testicular Tumors. Frontiers in endocrinology, 530. https://doi.org/10.3389/fendo.2014.00030
Chimento, Adele, et al. "GPER Signaling in Spermatogenesis and Testicular Tumors." Frontiers in endocrinology vol. 5 (2014): 30. doi: https://doi.org/10.3389/fendo.2014.00030
Chimento A, Sirianni R, Casaburi I, Pezzi V. GPER Signaling in Spermatogenesis and Testicular Tumors. Front Endocrinol (Lausanne). 2014 Mar 06;5:30. doi: 10.3389/fendo.2014.00030. eCollection 2014. PMID: 24639669; PMCID: PMC3944538.
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The gut-brain-axis as a target to treat stress-induced obesity.

Frontiers in endocrinology

Lee CY, Abizaid A.
PMID: 25101055
Front Endocrinol (Lausanne). 2014 Jul 18;5:117. doi: 10.3389/fendo.2014.00117. eCollection 2014.

No abstract available.

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Lee CY, Abizaid A. The gut-brain-axis as a target to treat stress-induced obesity. Front Endocrinol (Lausanne). 2014;5:117doi: 10.3389/fendo.2014.00117.
Lee, C. Y., & Abizaid, A. (2014). The gut-brain-axis as a target to treat stress-induced obesity. Frontiers in endocrinology, 5117. https://doi.org/10.3389/fendo.2014.00117
Lee, Chooi Yeng, and Abizaid, Alfonso. "The gut-brain-axis as a target to treat stress-induced obesity." Frontiers in endocrinology vol. 5 (2014): 117. doi: https://doi.org/10.3389/fendo.2014.00117
Lee CY, Abizaid A. The gut-brain-axis as a target to treat stress-induced obesity. Front Endocrinol (Lausanne). 2014 Jul 18;5:117. doi: 10.3389/fendo.2014.00117. eCollection 2014. PMID: 25101055; PMCID: PMC4102906.
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The Underexploited Role of Non-Coding RNAs in Lysosomal Storage Diseases.

Frontiers in endocrinology

Queiroz MT, Pereira VG, do Nascimento CC, D'Almeida V.
PMID: 27708618
Front Endocrinol (Lausanne). 2016 Sep 21;7:133. doi: 10.3389/fendo.2016.00133. eCollection 2016.

Non-coding RNAs (ncRNAs) are a functional class of RNA involved in the regulation of several cellular processes which may modulate disease onset, progression, and prognosis. Lysosomal storage diseases (LSD) are a group of rare disorders caused by mutations of...

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Queiroz MT, Pereira VG, do Nascimento CC, et al. The Underexploited Role of Non-Coding RNAs in Lysosomal Storage Diseases. Front Endocrinol (Lausanne). 2016;7:133doi: 10.3389/fendo.2016.00133.
Queiroz, M. T., Pereira, V. G., do Nascimento, C. C., & D'Almeida, V. (2016). The Underexploited Role of Non-Coding RNAs in Lysosomal Storage Diseases. Frontiers in endocrinology, 7133. https://doi.org/10.3389/fendo.2016.00133
Queiroz, Matheus Trovão, et al. "The Underexploited Role of Non-Coding RNAs in Lysosomal Storage Diseases." Frontiers in endocrinology vol. 7 (2016): 133. doi: https://doi.org/10.3389/fendo.2016.00133
Queiroz MT, Pereira VG, do Nascimento CC, D'Almeida V. The Underexploited Role of Non-Coding RNAs in Lysosomal Storage Diseases. Front Endocrinol (Lausanne). 2016 Sep 21;7:133. doi: 10.3389/fendo.2016.00133. eCollection 2016. PMID: 27708618; PMCID: PMC5030823.
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A Novel Germline Mutation of KEAP1 (R483H) Associated with a Non-Toxic Multinodular Goiter.

Frontiers in endocrinology

Nishihara E, Hishinuma A, Kogai T, Takada N, Hirokawa M, Fukata S, Ito M, Yabuta T, Nishikawa M, Nakamura H, Amino N, Miyauchi A.
PMID: 27703446
Front Endocrinol (Lausanne). 2016 Sep 20;7:131. doi: 10.3389/fendo.2016.00131. eCollection 2016.

BACKGROUND: A germline mutation of PATIENT FINDINGS: We report a 47-year-old Japanese woman who presented with hyperthyroidism and a large multinodular goiter. The family history was notable for a paternal history of goiter. Graves' disease was diagnosed based on...

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Nishihara E, Hishinuma A, Kogai T, et al. A Novel Germline Mutation of KEAP1 (R483H) Associated with a Non-Toxic Multinodular Goiter. Front Endocrinol (Lausanne). 2016;7:131doi: 10.3389/fendo.2016.00131.
Nishihara, E., Hishinuma, A., Kogai, T., Takada, N., Hirokawa, M., Fukata, S., Ito, M., Yabuta, T., Nishikawa, M., Nakamura, H., Amino, N., & Miyauchi, A. (2016). A Novel Germline Mutation of KEAP1 (R483H) Associated with a Non-Toxic Multinodular Goiter. Frontiers in endocrinology, 7131. https://doi.org/10.3389/fendo.2016.00131
Nishihara, Eijun, et al. "A Novel Germline Mutation of KEAP1 (R483H) Associated with a Non-Toxic Multinodular Goiter." Frontiers in endocrinology vol. 7 (2016): 131. doi: https://doi.org/10.3389/fendo.2016.00131
Nishihara E, Hishinuma A, Kogai T, Takada N, Hirokawa M, Fukata S, Ito M, Yabuta T, Nishikawa M, Nakamura H, Amino N, Miyauchi A. A Novel Germline Mutation of KEAP1 (R483H) Associated with a Non-Toxic Multinodular Goiter. Front Endocrinol (Lausanne). 2016 Sep 20;7:131. doi: 10.3389/fendo.2016.00131. eCollection 2016. PMID: 27703446; PMCID: PMC5028897.
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Non-Mammalian Vertebrates: Distinct Models to Assess the Role of Ion Gradients in Energy Expenditure.

Frontiers in endocrinology

Geisler CE, Kentch KP, Renquist BJ.
PMID: 28919880
Front Endocrinol (Lausanne). 2017 Sep 01;8:224. doi: 10.3389/fendo.2017.00224. eCollection 2017.

Animals store metabolic energy as electrochemical gradients. At least 50% of mammalian energy is expended to maintain electrochemical gradients across the inner mitochondrial membrane (H

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Geisler CE, Kentch KP, Renquist BJ. Non-Mammalian Vertebrates: Distinct Models to Assess the Role of Ion Gradients in Energy Expenditure. Front Endocrinol (Lausanne). 2017;8:224doi: 10.3389/fendo.2017.00224.
Geisler, C. E., Kentch, K. P., & Renquist, B. J. (2017). Non-Mammalian Vertebrates: Distinct Models to Assess the Role of Ion Gradients in Energy Expenditure. Frontiers in endocrinology, 8224. https://doi.org/10.3389/fendo.2017.00224
Geisler, Caroline E, et al. "Non-Mammalian Vertebrates: Distinct Models to Assess the Role of Ion Gradients in Energy Expenditure." Frontiers in endocrinology vol. 8 (2017): 224. doi: https://doi.org/10.3389/fendo.2017.00224
Geisler CE, Kentch KP, Renquist BJ. Non-Mammalian Vertebrates: Distinct Models to Assess the Role of Ion Gradients in Energy Expenditure. Front Endocrinol (Lausanne). 2017 Sep 01;8:224. doi: 10.3389/fendo.2017.00224. eCollection 2017. PMID: 28919880; PMCID: PMC5585156.
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CCL2/Monocyte Chemoattractant Protein 1 and Parathyroid Hormone Action on Bone.

Frontiers in endocrinology

Siddiqui JA, Partridge NC.
PMID: 28424660
Front Endocrinol (Lausanne). 2017 Mar 29;8:49. doi: 10.3389/fendo.2017.00049. eCollection 2017.

Chemokines are small molecules that play a crucial role as chemoattractants for several cell types, and their components are associated with host immune responses and repair mechanisms. Chemokines selectively recruit monocytes, neutrophils, and lymphocytes and induce chemotaxis through the...

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Siddiqui JA, Partridge NC. CCL2/Monocyte Chemoattractant Protein 1 and Parathyroid Hormone Action on Bone. Front Endocrinol (Lausanne). 2017;8:49doi: 10.3389/fendo.2017.00049.
Siddiqui, J. A., & Partridge, N. C. (2017). CCL2/Monocyte Chemoattractant Protein 1 and Parathyroid Hormone Action on Bone. Frontiers in endocrinology, 849. https://doi.org/10.3389/fendo.2017.00049
Siddiqui, Jawed Akhtar, and Partridge, Nicola C. "CCL2/Monocyte Chemoattractant Protein 1 and Parathyroid Hormone Action on Bone." Frontiers in endocrinology vol. 8 (2017): 49. doi: https://doi.org/10.3389/fendo.2017.00049
Siddiqui JA, Partridge NC. CCL2/Monocyte Chemoattractant Protein 1 and Parathyroid Hormone Action on Bone. Front Endocrinol (Lausanne). 2017 Mar 29;8:49. doi: 10.3389/fendo.2017.00049. eCollection 2017. PMID: 28424660; PMCID: PMC5372820.
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Diabetic Neuropathy Evaluated by a Novel Device: Sural Nerve Conduction Is Associated with Glycemic Control and Ankle-Brachial Pressure Index in Japanese Patients with Diabetes.

Frontiers in endocrinology

Hamasaki H, Hamasaki Y.
PMID: 28861044
Front Endocrinol (Lausanne). 2017 Aug 15;8:203. doi: 10.3389/fendo.2017.00203. eCollection 2017.

BACKGROUND: Currently, no international diagnostic criteria for diabetic neuropathy (DN) have been established. Recently, a novel point-of-care sural nerve conduction device has been developed. We aimed to investigate associations between DN and clinical parameters related to the development and...

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Hamasaki H, Hamasaki Y. Diabetic Neuropathy Evaluated by a Novel Device: Sural Nerve Conduction Is Associated with Glycemic Control and Ankle-Brachial Pressure Index in Japanese Patients with Diabetes. Front Endocrinol (Lausanne). 2017;8:203doi: 10.3389/fendo.2017.00203.
Hamasaki, H., & Hamasaki, Y. (2017). Diabetic Neuropathy Evaluated by a Novel Device: Sural Nerve Conduction Is Associated with Glycemic Control and Ankle-Brachial Pressure Index in Japanese Patients with Diabetes. Frontiers in endocrinology, 8203. https://doi.org/10.3389/fendo.2017.00203
Hamasaki, Hidetaka, and Hamasaki, Yasuteru. "Diabetic Neuropathy Evaluated by a Novel Device: Sural Nerve Conduction Is Associated with Glycemic Control and Ankle-Brachial Pressure Index in Japanese Patients with Diabetes." Frontiers in endocrinology vol. 8 (2017): 203. doi: https://doi.org/10.3389/fendo.2017.00203
Hamasaki H, Hamasaki Y. Diabetic Neuropathy Evaluated by a Novel Device: Sural Nerve Conduction Is Associated with Glycemic Control and Ankle-Brachial Pressure Index in Japanese Patients with Diabetes. Front Endocrinol (Lausanne). 2017 Aug 15;8:203. doi: 10.3389/fendo.2017.00203. eCollection 2017. PMID: 28861044; PMCID: PMC5559425.
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Accuracy of a Rapid and Non-Invasive Method for the Assessment of Small Fiber Neuropathy Based on Measurement of Electrochemical Skin Conductances.

Frontiers in endocrinology

Bordier L, Dolz M, Monteiro L, Névoret ML, Calvet JH, Bauduceau B.
PMID: 26973597
Front Endocrinol (Lausanne). 2016 Feb 29;7:18. doi: 10.3389/fendo.2016.00018. eCollection 2016.

No abstract available.

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Bordier L, Dolz M, Monteiro L, et al. Accuracy of a Rapid and Non-Invasive Method for the Assessment of Small Fiber Neuropathy Based on Measurement of Electrochemical Skin Conductances. Front Endocrinol (Lausanne). 2016;7:18doi: 10.3389/fendo.2016.00018.
Bordier, L., Dolz, M., Monteiro, L., Névoret, M. L., Calvet, J. H., & Bauduceau, B. (2016). Accuracy of a Rapid and Non-Invasive Method for the Assessment of Small Fiber Neuropathy Based on Measurement of Electrochemical Skin Conductances. Frontiers in endocrinology, 718. https://doi.org/10.3389/fendo.2016.00018
Bordier, Lyse, et al. "Accuracy of a Rapid and Non-Invasive Method for the Assessment of Small Fiber Neuropathy Based on Measurement of Electrochemical Skin Conductances." Frontiers in endocrinology vol. 7 (2016): 18. doi: https://doi.org/10.3389/fendo.2016.00018
Bordier L, Dolz M, Monteiro L, Névoret ML, Calvet JH, Bauduceau B. Accuracy of a Rapid and Non-Invasive Method for the Assessment of Small Fiber Neuropathy Based on Measurement of Electrochemical Skin Conductances. Front Endocrinol (Lausanne). 2016 Feb 29;7:18. doi: 10.3389/fendo.2016.00018. eCollection 2016. PMID: 26973597; PMCID: PMC4770015.
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Browning and Graying: Novel Transcriptional Regulators of Brown and Beige Fat Tissues and Aging.

Frontiers in endocrinology

Mueller E.
PMID: 26973598
Front Endocrinol (Lausanne). 2016 Mar 02;7:19. doi: 10.3389/fendo.2016.00019. eCollection 2016.

Obesity represents a major risk factor for the development of a number of metabolic disorders, including cardiovascular disease and type 2 diabetes. Since the discovery that brown and beige fat cells exist in adult humans and contribute to energy...

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Mueller E. Browning and Graying: Novel Transcriptional Regulators of Brown and Beige Fat Tissues and Aging. Front Endocrinol (Lausanne). 2016;7:19doi: 10.3389/fendo.2016.00019.
Mueller, E. (2016). Browning and Graying: Novel Transcriptional Regulators of Brown and Beige Fat Tissues and Aging. Frontiers in endocrinology, 719. https://doi.org/10.3389/fendo.2016.00019
Mueller, Elisabetta. "Browning and Graying: Novel Transcriptional Regulators of Brown and Beige Fat Tissues and Aging." Frontiers in endocrinology vol. 7 (2016): 19. doi: https://doi.org/10.3389/fendo.2016.00019
Mueller E. Browning and Graying: Novel Transcriptional Regulators of Brown and Beige Fat Tissues and Aging. Front Endocrinol (Lausanne). 2016 Mar 02;7:19. doi: 10.3389/fendo.2016.00019. eCollection 2016. PMID: 26973598; PMCID: PMC4773441.
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Cancer Stem Cells in the Thyroid.

Frontiers in endocrinology

Nagayama Y, Shimamura M, Mitsutake N.
PMID: 26973599
Front Endocrinol (Lausanne). 2016 Feb 29;7:20. doi: 10.3389/fendo.2016.00020. eCollection 2016.

The cancer stem cell (CSC) model posits that CSCs are a small, biologically distinct subpopulation of cancer cells in each tumor that have self-renewal and multi-lineage potential, and are critical for cancer initiation, metastasis, recurrence, and therapy-resistance. Numerous studies...

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Nagayama Y, Shimamura M, Mitsutake N. Cancer Stem Cells in the Thyroid. Front Endocrinol (Lausanne). 2016;7:20doi: 10.3389/fendo.2016.00020.
Nagayama, Y., Shimamura, M., & Mitsutake, N. (2016). Cancer Stem Cells in the Thyroid. Frontiers in endocrinology, 720. https://doi.org/10.3389/fendo.2016.00020
Nagayama, Yuji, et al. "Cancer Stem Cells in the Thyroid." Frontiers in endocrinology vol. 7 (2016): 20. doi: https://doi.org/10.3389/fendo.2016.00020
Nagayama Y, Shimamura M, Mitsutake N. Cancer Stem Cells in the Thyroid. Front Endocrinol (Lausanne). 2016 Feb 29;7:20. doi: 10.3389/fendo.2016.00020. eCollection 2016. PMID: 26973599; PMCID: PMC4770029.
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Severe Hypothyroidism due to the Loss of Therapeutic Efficacy of l-Thyroxine in a Patient with Esophageal Complication Associated with Systemic Sclerosis.

Frontiers in endocrinology

Lobasso A, Nappi L, Barbieri L, Peirce C, Ippolito S, Arpaia D, Rossi FW, de Paulis A, Biondi B.
PMID: 28979239
Front Endocrinol (Lausanne). 2017 Sep 20;8:241. doi: 10.3389/fendo.2017.00241. eCollection 2017.

BACKGROUND: Thyroid function abnormalities and thyroid autoantibodies have been frequently described in patients with systemic autoimmune diseases as systemic sclerosis (SSc). Serum TSH levels are higher in SSc patients with more severe skin diseases and a worse modified Rodnan...

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Lobasso A, Nappi L, Barbieri L, et al. Severe Hypothyroidism due to the Loss of Therapeutic Efficacy of l-Thyroxine in a Patient with Esophageal Complication Associated with Systemic Sclerosis. Front Endocrinol (Lausanne). 2017;8:241doi: 10.3389/fendo.2017.00241.
Lobasso, A., Nappi, L., Barbieri, L., Peirce, C., Ippolito, S., Arpaia, D., Rossi, F. W., de Paulis, A., & Biondi, B. (2017). Severe Hypothyroidism due to the Loss of Therapeutic Efficacy of l-Thyroxine in a Patient with Esophageal Complication Associated with Systemic Sclerosis. Frontiers in endocrinology, 8241. https://doi.org/10.3389/fendo.2017.00241
Lobasso, Antonio, et al. "Severe Hypothyroidism due to the Loss of Therapeutic Efficacy of l-Thyroxine in a Patient with Esophageal Complication Associated with Systemic Sclerosis." Frontiers in endocrinology vol. 8 (2017): 241. doi: https://doi.org/10.3389/fendo.2017.00241
Lobasso A, Nappi L, Barbieri L, Peirce C, Ippolito S, Arpaia D, Rossi FW, de Paulis A, Biondi B. Severe Hypothyroidism due to the Loss of Therapeutic Efficacy of l-Thyroxine in a Patient with Esophageal Complication Associated with Systemic Sclerosis. Front Endocrinol (Lausanne). 2017 Sep 20;8:241. doi: 10.3389/fendo.2017.00241. eCollection 2017. PMID: 28979239; PMCID: PMC5611405.
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Vanadate Impedes Adipogenesis in Mesenchymal Stem Cells Derived from Different Depots within Bone.

Frontiers in endocrinology

Jacobs FA, Sadie-Van Gijsen H, van de Vyver M, Ferris WF.
PMID: 27536268
Front Endocrinol (Lausanne). 2016 Aug 03;7:108. doi: 10.3389/fendo.2016.00108. eCollection 2016.

Glucocorticoid-induced osteoporosis (GIO) is associated with an increase in bone marrow adiposity, which skews the differentiation of mesenchymal stem cell (MSC) progenitors away from osteoblastogenesis and toward adipogenesis. We have previously found that vanadate, a non-specific protein tyrosine phosphatase...

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Jacobs FA, Sadie-Van Gijsen H, van de Vyver M, et al. Vanadate Impedes Adipogenesis in Mesenchymal Stem Cells Derived from Different Depots within Bone. Front Endocrinol (Lausanne). 2016;7:108doi: 10.3389/fendo.2016.00108.
Jacobs, F. A., Sadie-Van Gijsen, H., van de Vyver, M., & Ferris, W. F. (2016). Vanadate Impedes Adipogenesis in Mesenchymal Stem Cells Derived from Different Depots within Bone. Frontiers in endocrinology, 7108. https://doi.org/10.3389/fendo.2016.00108
Jacobs, Frans Alexander, et al. "Vanadate Impedes Adipogenesis in Mesenchymal Stem Cells Derived from Different Depots within Bone." Frontiers in endocrinology vol. 7 (2016): 108. doi: https://doi.org/10.3389/fendo.2016.00108
Jacobs FA, Sadie-Van Gijsen H, van de Vyver M, Ferris WF. Vanadate Impedes Adipogenesis in Mesenchymal Stem Cells Derived from Different Depots within Bone. Front Endocrinol (Lausanne). 2016 Aug 03;7:108. doi: 10.3389/fendo.2016.00108. eCollection 2016. PMID: 27536268; PMCID: PMC4971437.
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Showing 1 to 12 of 6108 entries